Skip to content
Help us to help more people

رنح فريدرايخ > المنشورات

Causative role of oxidative stress in a Drosophila model of Friedreich ataxia.

https://www.ncbi.nlm.nih.gov/pubmed/17167074
بواسطة Jose Vicente
الباحث

Friedreich ataxia: an update on animal models, frataxin function and therapies.

https://www.ncbi.nlm.nih.gov/pubmed/20225031
بواسطة Jose Vicente
الباحث

Overexpression of human and fly frataxins in Drosophila provokes deleterious effects at biochemical, physiological and developmental levels.

https://www.ncbi.nlm.nih.gov/pubmed/21779322
بواسطة Jose Vicente
الباحث

Deferiprone and idebenone rescue frataxin depletion phenotypes in a Drosophila model of Friedreich's ataxia.

https://www.ncbi.nlm.nih.gov/pubmed/23542074
بواسطة Jose Vicente
الباحث

TORC1 Inhibition by Rapamycin Promotes Antioxidant Defences in a Drosophila Model of Friedreich's Ataxia.

https://www.ncbi.nlm.nih.gov/pubmed/26158631
بواسطة Jose Vicente
الباحث

Metal Homeostasis Regulators Suppress FRDA Phenotypes in a Drosophila Model of the Disease.

https://www.ncbi.nlm.nih.gov/pubmed/27433942
بواسطة Jose Vicente
الباحث