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Friedreich ataxie > Publikace

Causative role of oxidative stress in a Drosophila model of Friedreich ataxia.

https://www.ncbi.nlm.nih.gov/pubmed/17167074
podle Jose Vicente
Vědecký pracovník

Friedreich ataxia: an update on animal models, frataxin function and therapies.

https://www.ncbi.nlm.nih.gov/pubmed/20225031
podle Jose Vicente
Vědecký pracovník

Overexpression of human and fly frataxins in Drosophila provokes deleterious effects at biochemical, physiological and developmental levels.

https://www.ncbi.nlm.nih.gov/pubmed/21779322
podle Jose Vicente
Vědecký pracovník

Deferiprone and idebenone rescue frataxin depletion phenotypes in a Drosophila model of Friedreich's ataxia.

https://www.ncbi.nlm.nih.gov/pubmed/23542074
podle Jose Vicente
Vědecký pracovník

TORC1 Inhibition by Rapamycin Promotes Antioxidant Defences in a Drosophila Model of Friedreich's Ataxia.

https://www.ncbi.nlm.nih.gov/pubmed/26158631
podle Jose Vicente
Vědecký pracovník

Metal Homeostasis Regulators Suppress FRDA Phenotypes in a Drosophila Model of the Disease.

https://www.ncbi.nlm.nih.gov/pubmed/27433942
podle Jose Vicente
Vědecký pracovník