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Friedreich共济失调 > 动态

Causative role of oxidative stress in a Drosophila model of Friedreich ataxia.

https://www.ncbi.nlm.nih.gov/pubmed/17167074
通过 Jose Vicente
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Friedreich ataxia: an update on animal models, frataxin function and therapies.

https://www.ncbi.nlm.nih.gov/pubmed/20225031
通过 Jose Vicente
研究者

Overexpression of human and fly frataxins in Drosophila provokes deleterious effects at biochemical, physiological and developmental levels.

https://www.ncbi.nlm.nih.gov/pubmed/21779322
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研究者

Deferiprone and idebenone rescue frataxin depletion phenotypes in a Drosophila model of Friedreich's ataxia.

https://www.ncbi.nlm.nih.gov/pubmed/23542074
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TORC1 Inhibition by Rapamycin Promotes Antioxidant Defences in a Drosophila Model of Friedreich's Ataxia.

https://www.ncbi.nlm.nih.gov/pubmed/26158631
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Metal Homeostasis Regulators Suppress FRDA Phenotypes in a Drosophila Model of the Disease.

https://www.ncbi.nlm.nih.gov/pubmed/27433942
通过 Jose Vicente
研究者